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Anti-ulcer with Natural DGL Licorice Vitamin Supplements

With peptic ulcers on the rise, medical treatment treaty has changed radically in recent years. Now available a wide variety of antacids, antihistamines and proton pump inhibitors, which cover only the symptoms but does not address the real cause of peptic ulcer. In this article we will discuss what a peptic ulcer is its symptoms and causes. We will also see how ulcers are currently treated and compare the side effects of conventional medicine to DGL licorice, and how to take DGL Licorice and where You can find this item for sale that match.

Peptic ulcers form in the lining of the stomach, small intestine and esophagus. These areas erode stomach ulcers, the acid if unchecked can be fatal. Ulcers in the stomach are called gastric ulcers and ulcers in the intestine are called duodenal ulcers. Peptic ulcers are a chronic problem, and who go into remission, then activate over time through of a life.

Peptic ulcers are very common in the U.S., where one in ten people develop ulcers for life. Ulcers are sores the most common and easiest to heal. Stomach ulcers usually return more often with more discomfort. Individuals with peptic ulcers may have pain of little or no pain. Others may experience burning and pain associated with cramps and back a few days to several weeks. Most people experience pain two to three hours after eating or symptoms can explode in the middle of the night. Most people can reduce pain by eating food. Other symptoms may be weight loss, lack of appetite, bloating, belching, nausea and vomiting. (1.2)

During the last two decades there has been a radical change in what doctors that causes peptic ulcers. In the past, ulcers were attributed to stress, spicy foods, alcohol consumption and gastric acid production but now researchers have discovered most ulcers are the cause is a bacterial infection. Helicobacter pylori (H. pylori) represents the majority of cases of ulcer in the United States.

Some over the counter and prescription painkillers can also cause ulcers. Drugs known as anti-inflammatory drugs (NSAIDs) can also cause peptic ulcers. These are more common than you can know that ibuprofen is in Motrin and Advil.

You may wonder how you know if you have this bacterium H. pylori in the body. Current technology allows three modes of control, blood, breath test and tissue analysis. Blood test is the way most commonly used detection. A breath test was used after treatment to kill the bacteria to determine whether the treatment worked.

Normally 20% of Americans under age 40 and 50% of Americans over age 60 have the bacteria H. pylori. Even if some people have the bacteria, which do not come with ulcers so researchers are studying why this occurs in some people. Researchers have also discovered some people do not come down with ulcers while taking these medications for long periods of time led researchers to consider other factors in the environment can be handy device with the development of ulcers. (3.4)

In the medical community today treat patients H. pylori with triple therapy theory ulcer. Patients are prescribed antibiotics, a powerful anti-acid and stomach protectors. Antibiotics usually kill bacteria if this is the cause of the ulcer. Some of these drugs are acid-suppression of histamine-2 receptor antagonists Tagamet, Zantac, Pepcid, and inhibitors of proton pump Prevacid and Prilosec.

It is important to find a licensed health care professional if you believe you have an ulcer. Ulcers can erode the stomach lining and cause fatal bleeding and infections such as peritonitis. Despite the risk of unwanted side effects of medication, is important to consult a doctor before taking matters into their own hands. There are natural supplements that work well with medications that can speed the healing of the ulcer. In some cases, milder conditions prior ulcer can be treated with more natural solutions first if your health professional license so decides.

There are natural alternatives that work and for the treatment of an ulcer and can be used in combination with antibiotics and other prescription drugs. Licorice Root Deglycrrhizinated specifically liquorice (DGL) may be a good natural complement to other therapeutic measures recommended by your health care procession. Researchers studied LSD in the treatment of gastric ulcers and duodenal ulcers. (5-12)

DGL actually addresses the underlying problem causing ulcers instead of hiding symptoms by reducing gastric acidity. DGL address the factors underlying the promotion of natural defense mechanisms of our body is already under way to prevent ulcers. DGL stimulates the quantity and quality of the protective substance that lines the stomach and intestinal tract. (10.13)

DGL is a special extract Liquorice some deleted items. The glycyrrhizin molecules have been removed from licorice which is associated with high blood pressure and low potassium levels. Sodium was eliminated of the DGL licorice which will help prevent water retention. The long-term use of licorice root can have undesirable side effects such as water retention, Flavonoids of high blood pressure and low potassium DGL has all the components removed and what remains are very beneficial biologically active.

In 1982, researchers reported DGL was as effective as Tagamet in curing gastric ulcers. (14) The same year, DGL has also been reported to be as good as Zantac. (6) Licorice root extract in the form of DGL stimulates the release of secretin. Secretin has a protective effect on the gastric mucosa. By stimulating the natural release of endogenous secretin body, the body can not rebuild the gastric or intestinal mucosal damage. (15)

In the past, antacids were the number one prescription drug for ulcers, but have since been replaced by inhibitors of proton pump. Antacids do have side effects unpleasant in the gut, for example, aluminum hydroxide promotes constipation and other anti-acids, such as magnesium hydroxide promote diarrhea. Antacids reduce stomach acid and may reduce the absorption of vitamins and medications.

Compared with other drugs such as antacids DGL, Tagamet, Zantac, Prilosec and Prevacid all have side effects that DGL has none. Antacids such as magnesium hydroxide, aluminum hydroxide, carbonate calcium and aluminum (Maalox, Mylanta, Gelusil and Tums) can cause rebound hyperacidity, a condition in which the body creates more acid in response to the disabling artificial gastric acid. Antacids can also have bowel changes such as diarrhea or constipation and possible drug interactions. Due the high sodium content of antacids deterioration of persons with disabilities should consult a doctor before use. Tagamet, Zantac, Prilosec and Prevacid have the following effects side, respectively. Tagamet can cause dizziness, drowsiness, headache, confusion, hallucinations, diarrhea, and impotence in men. (16) Zantac may cause headaches, constipation, diarrhea, nausea, abdominal pain and rash. (17) and Prevacid Prilosec can cause headaches, dizziness, diarrhea, pain abdominal pain, nausea, vomiting, constipation, upper respiratory symptoms. (18.19)

DGL has none of the above side effects and is easy use. DGL should be taken 20 minutes before each meal at 760 or 1520 mg. The best way to consume DGL is to chew and mix with saliva in the mouth. The compounds of saliva in the mouth help stimulate growth and regeneration of stomach and intestinal cells. Use DGL 8 to 16 weeks or for as long as your provider recommends health care. In conclusion DGL can help improve the integrity of the lining of the stomach and intestines and help the recovery of nasty ulcers. DGL AIDS and stomach can be found others in your health food store or Internet.

References:

1. Peptic ulcer. In: Guyton AC, Hall JE. Medical physiology. Philadelphia, Pa: WB Saunders Company, 1998:846-847.

2. Peptic ulcer. In: Porth CM. Pathophysiology: Concepts of Altered States of Health. 5th ed. Philadelphia, Pa: Lippincott, 1998: 725-728.

3. Dajani EZ, Klamut MJ. New therapeutic approaches against gastric and duodenal ulcers: an update. Expert Opin Investig Drugs. 2000, 9:1537-1544.

4. Cappell MS, Schein JR. Diagnosis and treatment of anti-inflammatory drugs associated with increased gastrointestinal toxicity. Gastroenterol Clin North Am 2000, 29:97-124.

5. Engqvist A, von Feilitzen F, Pyk E, Reichard H. Double-blind, DGL liqourice gastric ulcer. Gut. 1973, 14:711-715.

6. L. DGL Licorice Glick of peptic ulcer. Lancet. 1982, 9:817.

7. Bardhan KD, Cumberland DC, Dixon RA, Holdsworth CD. Deglycyrrhisinated trials Liqourice gastric ulcer. Gut. 1978, 19:779-782.

8. Balakrishnan V Pillai MV, Raveebdran PM, Nair CS. Liqourice Deglycrrhizinated in the treatment of chronic duodenal ulcer. J Assoc Physicians India. 1978, 26:811-814.

9. Rees WDW, Rhodes J, Wright JE, Stamford IF, A. Effect of injury Bennett DGL licorice in gastric mucosa of aspirin. Scand J Gastroenterol. 1979, 14:605-607.

10. Tewari SN Wilson AK. Licorice Deglycrrhizinated in the ulcer duodenal. Professional. 1973; 210:820-823.

11. Abrahamsson H, Dotevall G. Pharmacological and clinical aspects of some medicines used to treat ulcer ulcer. Scand J Gastroenterol. 1979, 55:117-120.

12. Bardnan KD, Cumberland DC, Dixon RA, Holdsworth CD. Minutes: liqourice Deglycrrhizinated in gastric ulcer: a controlled double-blind. Gut. 1976, 17:397.

13. Morgan AG, Pacsoo C, McAdam WAF. Maintenance therapy: a comparison between two years collapsed-S cimetidine and treatment to prevent recurrence of symptoms of gastric ulcer. Gut. 1985, 26:599-602.

14. Morgan AG, McAdam WAF, Pacsoo C, Darnborough A. Comparison of cimetidine and handed-S in the treatment of gastric ulcers, and maintenance therapy. Gut. 1982, 23:545-551.

15. Takeuchi T, Shiratori K, Watanabe S, Chang JH, Moriyoshi Y, Shimizu K. Secretin as a potential mediator agents antiulceractions mucosal protection. J Clin Gastroenterol. 1991, 13:83-87.

16. Cimetidine. In: Physician's Desk Reference. "54th ed. Montvale, NJ: Medical Economics Company, Inc., 2000:3043-3046.

17. Ranitidine. Ibid. pp. 1310-1312.

18. Omeprazole. Ibid. pp. 617-621.

19. Lansoprazole. Ibid. pp. 3105-3110.

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